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Woof was a very special friend.
In loving memory.
By (and for) Marcy Rauch, written to the deafdog list and used with her permission
It is with enormous sadness that I say goodbye to a legend. Woof left my life at 7:45 pm last night (7.16.02) at 15 years and 12 days of age. Woof was not the kind of legend that many of you think about. She was not a top show dog or a movie star, but she was a dog that comes along once in a lifetime. For those of you who had the joy of meeting her, you got to see the beauty of spirit from within. Woof took life head-on with happiness and determination. She shattered the boundaries of her silent world by teaching so many what she could do. She learned agility at 12 years young, herded sheep for the first time at 11. She had been a quadriplegic 8 years ago from tick paralysis, but learned how to stand again. When all the other dogs missed a biscuit that had fallen Woof saw it and with sheer determination, forced herself up to get that biscuit. She taught children that deaf dogs were special and even brought tears to the eyes of a deaf child who felt alone in her plight. This past year, Woof was confined to a wheelchair. There, too, she showed her mettle by hiking and swimming and showing no defeat. She got to have a wheelchair race with a little boy with spina bifuda (of course we let him win by a hair) and her last outing on Saturday, she shared what she had left with a young man with cerebral palsy and brought a big smile to Joey and his family. This IS what legends are made of. People have asked how I could keep her going all this time and my answer was that as long as she had a smile on her face, I would do whatever it took to keep her going. Well, she stopped smiling on Sunday night, so we spent the next two days together and then I got to return the thousands of gifts she gave me in our 15 years together by giving her the greatest gift I could I helped her leave the body that had given up. Sleep well my friend. Your spirit and strength will go on in my heart forever. Marcy, Woofangel, Bandit, Echo and the cat trio top ![]()
A canine 'wheel chair' gave Woof the mobility she enjoyed.
PROGRESSIVE REAR LIMB WEAKNESS OR PARALYSIS
By the Southern California Veterinary Referral Group,
(reprinted here with permission of Dr Gilbert Velasquez, Administrator)
Degenerative myelopathy (DM) was first described as a specific, progressive, degenerative neurologic disease in 1973. Since then, much has been done to understand the processes involved in this disease and in the treatment of DM. The age at onset is 5 to 14 years. Cases have been reported in almost all large breeds of dogs, with the disease appearing with relative frequency only in the German Shepherd. This suggests that there is a predisposition for German Shepherd dogs to develop DM. The work presented here and by others on the nature of DM has been performed on the German Shepherd, the Belgian Shepherd and the Old English Sheepdog. It is currently not known whether the same condition exists in all breeds of dogs or if subtypes of the condition occur. The classic presentation is a painless, slowly progressive rear limb weakness or paralysis. There may be discomfort due to arthritis in the hip or lower lumbar (lower back) area, but this usually improves with activity. Over days, weeks or months the patient becomes progressively weaker as is evidenced by "shuffling" of the rear limbs and lack of coordination. Finally, full paralysis coupled with fecal and urinary incontinence develops.
DIAGNOSIS
A diagnosis of DM is made based on a history of progressive spinal ataxia and weakness that may be waxing or steadily progressive. This tentative diagnosis is supported by the neurological findings of widespread thoracolumbar (back) spinal cord dysfunction. Laboratory findings are generally normal except for an elevated cerebral spinal fluid (CSF) protein. A Myelogram (a contrast dye study of the spine) must be performed to differentiate DM from Disc Disease, Tumors, and other Progressive Neurologic Diseases. The striking feature common to each syndrome is the reduction of rear limb support. Current research points toward an immune mediated pathogenesis of DM. This provides a logical explanation for the presence of immune abnormalities in German Shepherd dogs with DM. It is hoped that work in the area with the antigens present in the immune-complexes will lead to a major breakthrough in our understanding of DM and to an early blood test for this condition.
TREATMENT
The treatment of DM involves four basic approaches: 1) Exercise 2) Supportive Measures 3) Medication 4) Minimization of Stress Exercise is extremely important in maintaining the well-being of affected dogs, maximizing muscle tone, and maintaining good circulation and conditioning. This is best achieved by an increasing schedule of alternate day exercise. Since may dogs have lost muscle tone prior to their diagnosis, it is important to gradually build their level of activity. The goal is to do aerobic exercise for 30 minutes twice a week and 1 hour once a week. This can begin with walking and gradually build to a faster pace. While not all patients can reach this goal, it is important to strive to do so. Running loose on the owners property is not adequate. Regular periods of programmed, continuous exercise are the most important. It is equally important that the patient with DM be allowed to rest on the day exercise is not scheduled. This will allow strained muscles and tendons to heal and will increase muscle strength. The dogs do not have to be confined, but merely discouraged from any strenuous exercise on these days of rest. It will be the consistent, controlled building of muscle tone through exercise that will help to delay the progression of DM. Vitamin support may be useful in delaying DM symptoms. Some recommend patients receive 200 IU of Vitamin E daily and one high potency B vitamin (B-1) every 12 hours. Synthetic vitamins are cheaper and just as effective as "natural" vitamins in this regard. No other supplementation of a balanced diet is needed or indicated in the treatment of DM. Because Vitamin E, at the levels recommended, is a non-steroidal anti-inflammatory agent, concurrent use of aspirin-like drugs is not recommended. Should aspirin-like drugs be required to treat arthritis in DM patients, the daily Vitamin E supplementation is reduced to 100 IU daily. One medication may prevent progression or result in clinical remission of DM in over 15 to 20% of the patients. This medication is aminocaproic acid (Amicar). It is given orally at 500mg every eight hours. Now that the "pill" form of medication has become expensive, we recommend giving aminocaproic acid as a solution, using the generic product. The product, while designed for injection, can be mixed with a vitamin-mineral supplement to provide a palatable solution for oral usage. The only side effects that have been attributed to aminocaproic acid have been occasional gastro-intestinal irritation. This prevents a problem in only a few patients, usually those who have pre-existing GI problems. In some dogs, vitamin supplements can cause excessive flatulence, necessitating that they receive the aminocaproic acid solution undiluted with the vitamin-mineral solution.
PROGNOSIS
DM progresses at different rates in each affected animal. Stress plays a role in its advancement. Minimizing stressful situations is important when possible. While anesthesia does not appear to cause problem with DM, even minor invasive surgical procedures can result in a marked increase in clinical signs of DM. Therefore, we recommend caution in considering major surgery in patients with DM. Unfortunately, the worsening caused by surgical stress can be irreversible. Woof: In Memory of a Special Dog Copyright © 2002 - 2005 by the Marcy Rauch, Southern California Veterinary Referral Group and AfterHours Australian Shepherds. No reproduction of any kind, of any of the material on this site, is permitted without the written permission of AfterHours Australian Shepherds, or in the case of previously published articles, the indicated author. |
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